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Some fatty acids, such as palmitic acid, can activate (phosphorylize) IRS-1 which causes
negative feedback to the insulin receptor and desensitizes muscle cells to insulinstimulated glucose uptake; curcumin appears to prevent this from occurring.[167] This
effect is shared by Green Tea Catechins.[167] Improvements in this mechanism of insulin
resistance have been seen in vivo with dose-dependent oral doses of curcumin at 50, 150,
and 250mg/kg bodyweight.[168] AMPK activation appears to be a key intermediate in
these effects.[168][102] Beyond acting upon IRS, curcumin may also increase glucose
uptake into skeletal muscles by acting on muscarinic acetylcholine receptors and then
through PLC and PI3K.[169]
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